West Nile disease

What is it West Nile disease?

West Nile virus is spread through the bite of an infected mosquito. The mosquito can become infected after biting an infected bird and this can then be transmitted from the bird to humans.

There is no evidence to suggest that West Nile virus can be spread from person to person or animal to person.

Where am I at risk?

The West Nile virus was first discovered in the West Nile area of Uganda in 1937.

It is prevalent in North America, where two thirds of the East coast states have had cases. There has even been a case in California.

There have also been cases in Israel, Africa and Asia and parts of Eastern Europe.

There was a UK case of a bird with the antibodies of the West Nile virus, but there have been no human cases.

What are the symptoms?

The patient can incubate the disease for up to a fortnight after being bitten.

Many of the people who become infected will have either no symptoms or mild flu-like symptoms, which last a couple of days.

These can range from fever, headache and body aches to skin rashes on the trunk of the body and swollen lymph glands.

But more serious cases, less than 1% of those who become infected, can result in severe illness such as West Nile encephalitis or meningitis and sometimes lead to death.

Out of about 1,000 people who became infected in Europe recently there were about 40 deaths.

How do I protect myself?

Scientists have developed a vaccine for the disease and it was due to start human trials in late 2002.

But as yet there is no vaccine for travellers to use so the best way to protect yourself is to avoid being bitten by a mosquito.

The mosquitoes which carry this disease bite at dawn, dusk and early evening, so if you are travelling to an area where the disease is prevalent you should either stay indoors, or wear long sleeved shirts and long-trousers and wear insect repellent.

People travelling with young children should ensure that they use a specially prepared insect repellent, rather than the adult version.

Leishmaniasis

What is it?

Leishmaniasis is a parasitic disease spread through bites from sandflies.

It affects about 12 million people globally, most of them in south or central America, Africa and the Middle East.

There are three sorts of leishmaniasis, which vary in severity.

The most dangerous is "visceral leishmaniasis", which is also known as black fever. This is nearly always fatal if it is left untreated.

It spreads into the spleen, bone marrow and liver and attacks and destroys the immune system.

Another type, "mucocutaneous leishmaniasis", produces disfiguring lesions which destroy the mucous membranes of the nose, mouth and throat.

"Cutaneous leishmaniasis" tends to cause sores on the skin. Some people have had up to 100 and these can vary in size from 2cm to 3cm in diameter.

How can I avoid it?

As the disease is spread by the sandfly the best way to avoid it, is to avoid getting bitten.

The sandfly bites mainly at night so travellers should use a good insect repellent, they should also sleep under insect nets with a very fine mesh.

Leishmaniasis is prevalent in about 90 tropical and subtropical countries around the world.

What do I do if I get it?

Anyone suffering from the symptoms of leishmaniasis, who has just returned from an area in which it is prevalent, should contact their GP immediately.

It is vital that people going to their doctor tell him where they have been travelling so that he can make an accurate diagnosis.

What is the treatment?

The treatment involves a series of about 30 days worth of injections, using a toxic drug to kill the parasite. The patient has to stay in hospital for the duration of their treatment.

Health in Somalia

The average Somali is struggling hard to receive a human treatment in the African Horn. Life in this part of the world has not been easy over the last decade. War, internal rebellion and economic breakdowns have left many thousands displaced. Children and women have come to bear the maximum brunt of these unsolicited evils. Malnutrition, disablement and fatal diseases are all too, very common. With the current war scene showing no signs of ending early, there are serious doubts raised over the sustenance of human life here.

[Undernourished children everywhere in Somalia]

Meanwhile, the UNICEF (United Nations Children’s Fund) has announced plans to immunize about 100,000 internally displaced people, comprising women and children.

A New Syndrome That Promotes-Hyperlipidemia, Hypertension & Other Fatal Diseases is Found

The family of herpes virus now includes 8 kinds of viruses. HHV2 (HSV) & HHV6 (CMV) are highly Neuroinvasive. Due to the Neuro invasive nature of these viruses, the immuno compromised neurons become vulnerable to the viral RNA’s invasion. Post infection, a fraction of neurons is left with viral DNA present in Episomal form.
During the Latent Phase of the virus, productive cycle genes generally transcribe but functionally quiescent and only the latency associated transcription (LAT) is expressed. The vulnerable neurons are invaded by the viral RNA’s and LAT transcription continues for life.
Therefore, the respective controlled organs by these effected neurons experience the disorder in its homeostasis and become dysfunctional over a period of time.
Up until the middle age, excessive heat exposure, internal injury, epinephrine shock, the autoimmune system combats this invasion and suppresses the disease process. As the immune system compromises, the neuronal defense lowers down.
The molecular composition of the enzymes or stimuli produced by the viral RNA ridden neurons is altered from its original state and carries a grave influence of the invading RNA’s.
The neuron and its carrier motor nerve fiber experience this pathology to the end tissue and cells.

This gives rise to the cellular, tissue and organ dysfunctions. Since the specificity of the dysfunction roots to the Neuro Invasive disease that jeopardizes the neurological integrity of the controlled organ, the secondary disease come into play.
This is the “Sheikh’s Syndrome”.

Clinically it presents as an infection due to the autoimmune and neuro immune disorder. Antibodies against these Neuroinvasive viral RNA’s present in the blood quantatively demonstrates its chronicity of the infection as IgG. This proves the cytopathology.
Therefore, it is the syndrome (Sheikh’s Syndrome) which becomes the root cause of the following secondary diseases.



• ADD * Allergies* Alzheimer
• Amenorrhea * Anemia * Aneurysms
• Atherosclerosis * Asthma * Bell’s & Cerebral Palsy
• CAD * Cancer * CFS
• Cognitive Disorder * Chromosomal Disorder* Diabetes Mellitus
• Encephalitis * Endocrine Disorder * Gastroenteritis
• Gastric Ulcer * Gynecomastia * Hypertension
• Hypohemoglobinemia * Hyperlipidemia * Hypothalamic Dysf.
• Hypo/ hyperthyroidism * Impotence * Infertility
• IBS * Liver Dysf. * Meningitis
• Metabolic Disorder * M.S. * Neoplastic-Tumors
• Neuralgia * Neuropathy * Nephropathy
• Parkinson’s * Psychiatric disorder* Pituitary Dysfunctions
• Recurrence of Minor Infections
• Sudden Tachycardia * Schizophreni* Respiratory Failure
• Stroke * Teenage Drug Addiction
In today’s world, we are dealing with enormous health challenges that stem from these root causes. The motor neurons responsible for the liver control connected to the vagus nerve, partial or fully affected with this cytopathology, affect various dysfunctions of the liver. Hyperlipidemia is a prominent dysfunction of the liver; it tracks back to the viral RNA invasion of the controlling neurons and the motor nerve fiber in the vagus bundle of nerve fibers, cytopathologically substantiates this dysfunction.

When successfully combated the viral RNA invasion, a new world record of hyperlipidemia deterrence 82% reduction
in LDL-c in 10 consecutive days was set in.

Similarly, the hypertension’s syndrome “X” also has been found to carry out the same pathophysiology that invades the
cardiac center at the brain stem, influencing the cardiac nerve fiber of the vagus bundle of nerve fibers to the heart’s S.A.
Therefore the cardiac neuro network projects this pathology in to the cardiac perkinji fibers.
These nerve ending are not immune to this pathology. When perkinji fibers network is experiencing the lesions, the cardiac output is gravely influenced and hypertension sets in. Hypertension has been alleviated by deterring this pathology.