A New Syndrome That Promotes-Hyperlipidemia, Hypertension & Other Fatal Diseases is Found

The family of herpes virus now includes 8 kinds of viruses. HHV2 (HSV) & HHV6 (CMV) are highly Neuroinvasive. Due to the Neuro invasive nature of these viruses, the immuno compromised neurons become vulnerable to the viral RNA’s invasion. Post infection, a fraction of neurons is left with viral DNA present in Episomal form.
During the Latent Phase of the virus, productive cycle genes generally transcribe but functionally quiescent and only the latency associated transcription (LAT) is expressed. The vulnerable neurons are invaded by the viral RNA’s and LAT transcription continues for life.
Therefore, the respective controlled organs by these effected neurons experience the disorder in its homeostasis and become dysfunctional over a period of time.
Up until the middle age, excessive heat exposure, internal injury, epinephrine shock, the autoimmune system combats this invasion and suppresses the disease process. As the immune system compromises, the neuronal defense lowers down.
The molecular composition of the enzymes or stimuli produced by the viral RNA ridden neurons is altered from its original state and carries a grave influence of the invading RNA’s.
The neuron and its carrier motor nerve fiber experience this pathology to the end tissue and cells.

This gives rise to the cellular, tissue and organ dysfunctions. Since the specificity of the dysfunction roots to the Neuro Invasive disease that jeopardizes the neurological integrity of the controlled organ, the secondary disease come into play.
This is the “Sheikh’s Syndrome”.

Clinically it presents as an infection due to the autoimmune and neuro immune disorder. Antibodies against these Neuroinvasive viral RNA’s present in the blood quantatively demonstrates its chronicity of the infection as IgG. This proves the cytopathology.
Therefore, it is the syndrome (Sheikh’s Syndrome) which becomes the root cause of the following secondary diseases.



• ADD * Allergies* Alzheimer
• Amenorrhea * Anemia * Aneurysms
• Atherosclerosis * Asthma * Bell’s & Cerebral Palsy
• CAD * Cancer * CFS
• Cognitive Disorder * Chromosomal Disorder* Diabetes Mellitus
• Encephalitis * Endocrine Disorder * Gastroenteritis
• Gastric Ulcer * Gynecomastia * Hypertension
• Hypohemoglobinemia * Hyperlipidemia * Hypothalamic Dysf.
• Hypo/ hyperthyroidism * Impotence * Infertility
• IBS * Liver Dysf. * Meningitis
• Metabolic Disorder * M.S. * Neoplastic-Tumors
• Neuralgia * Neuropathy * Nephropathy
• Parkinson’s * Psychiatric disorder* Pituitary Dysfunctions
• Recurrence of Minor Infections
• Sudden Tachycardia * Schizophreni* Respiratory Failure
• Stroke * Teenage Drug Addiction
In today’s world, we are dealing with enormous health challenges that stem from these root causes. The motor neurons responsible for the liver control connected to the vagus nerve, partial or fully affected with this cytopathology, affect various dysfunctions of the liver. Hyperlipidemia is a prominent dysfunction of the liver; it tracks back to the viral RNA invasion of the controlling neurons and the motor nerve fiber in the vagus bundle of nerve fibers, cytopathologically substantiates this dysfunction.

When successfully combated the viral RNA invasion, a new world record of hyperlipidemia deterrence 82% reduction
in LDL-c in 10 consecutive days was set in.

Similarly, the hypertension’s syndrome “X” also has been found to carry out the same pathophysiology that invades the
cardiac center at the brain stem, influencing the cardiac nerve fiber of the vagus bundle of nerve fibers to the heart’s S.A.
Therefore the cardiac neuro network projects this pathology in to the cardiac perkinji fibers.
These nerve ending are not immune to this pathology. When perkinji fibers network is experiencing the lesions, the cardiac output is gravely influenced and hypertension sets in. Hypertension has been alleviated by deterring this pathology.